Clinical Neuropathology. Text and Color Atlas by Catherine Haberland MD

By Catherine Haberland MD

This full-color textual content and atlas, presents an entire evaluate of the structural changes of significant fearful procedure illnesses, besides their medical manifestations. Readers will comprehend the pursuits of the neuropathologic examination, the histologic response of neural tissue to illnesses, and the pathologic stipulations specific to the worried procedure. All themes are highlighted via gross and histologic photos of the mind and spinal wire, and all combine the latest medical reports.

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The cerebral cortex is normal or mildly atrophic. Hydrocephalus Ex Vacuo Other Forms of Hydrocephalus Normal Pressure Hydrocephalus Normal pressure hydrocephalus (NPH), a particular form of obstructive hydrocephalus, is distinguished by a progressive enlargement of the ventricles without clinical evidence of raised intracranial pressure. The CSF flow is obstructed in the subarachnoid space or at the arachnoid villi. In a few cases, the history discloses previous head trauma, meningitis, or subarachnoid hemorrhage; however, in a large percentage of cases, no cause is evident.

Transverse section reveals a large infarct in the right Ammon horn, hippocampus, and fusiform gyrus. The right fornix, the most prominent efferent connection to the mammillary body, is degenerated (wallerian degeneration). The right mammillary body, not included in the picture, is severely atrophic (trans-synaptic atrophy; myelin stain). olivary nuclei atrophy when the Purkinje cells in the contralateral cerebellar cortex have degenerated. Retrograde Trans-Synaptic Degeneration Dying-Back Degeneration Retrograde trans-synaptic degeneration develops in those neurons that project to neurons that have already degenerated.

A. Thrombotic occlusion of MCA in the Sylvian fissure causing a large infarct. B. Cholesterol embolus in a small leptomeningeal artery overlying a cortical infarct, hematoxylin-eosin (HE) C. Hemodynamically significant (greater than 75%) stenosis in a carotid endarterectomy specimen. 3 Pathologic forms of arteriolosclerosis. A. Fibrohyalinosis with mural thickening and luminal stenosis (HE), B. Mural lipid deposits (oil-red-O). C. Eccentric microatheroma (LFB-HE). D. Markedly thickened, structureless fibrotic wall and virtually nonexisting lumen (HE).

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