Ciba Foundation Symposium 168 - Functional Anatomy of the

Utilizing a multidisciplinary technique, it combines anatomical, mobile and physiological reviews through preeminent specialists. gains vital insights into the functionality of the hypothalamus.

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In response to perturbation of neural inputs, or any perturbation we care to make of the steroid environment, markers for Co-localization in neuroendocrine neurons 31 both peptides tend to respond in tandem, but those for vasopressin always exhibit the greater sensitivity. I continue to be perplexed by the fact that two peptides in the same cell that serve complementary functions appear to be regulated under all circumstances in the same way. Clarke: Paul, you showed us that after adrenalectomy there was increased expression of CRF and AVP in the PVN (Fig.

Specificity of the effect of steroid withdrawal on secretagogues for corticotropin. Fluorescence photomicrographs to compare the distribution of corticotropin-releasing factor (CRF), vasopressin (Vas), neurotensin (NT) and met-enkephalin (m-Enk) immunoreactivity at a similar level of the paraventricular nucleus in untreated (Normal), colchicine-treated (Colch) and adrenalectomized (ADX) rats. In the untreated animals, only magnocellular vasopressin cells are prominent. Colchicine treatment reveals the ability of parvocellular neurons to display CRF, m-Enk and NT immunostaining.

Histochemistry 84:201-205 Manzanares J , Lookingland KJ, Moore KE 1990 Atrial natriuretic peptide-induced suppression of basal and dehydration-induced vasopressin secretion is not mediated by hypothalamic tuberohypophysial or tuberoinfundibular dopaminergic neurons. Brain Res 527:103-108 Merighi A, Polak JM, Theodosis DT 1991 Ultrastructural visualization of glutamate and aspartate immunoreactivities in the rat dorsal t m, with special reference to the co-localization of glutamate, substance P and alcitonin gene-related peptide.

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