Bronchopulmonary Dysplasia, Volume 240 (Lung Biology in by Steven H. Abman

By Steven H. Abman

Rising remedies for BPD - resembling Inhaled Nitrous Oxide, diet A, Antioxidant options, and low-dose steroid treatment

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Extra info for Bronchopulmonary Dysplasia, Volume 240 (Lung Biology in Health and Disease)

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1 is expressed in foregut endoderm-derived epithelial cells including developing lungs, thyroid, and pituitary, as well as in some restricted regions of fetal brain (56,57). 1À/À mice suffer severe impairment in branching morphogenesis of the lung and tracheoesophageal septum formation. 1 knockout mice, which indicates that lung development is arrested at a very early stage (58,59). 1 is expressed in the proximal and distal airway epithelia and, at later stages of lung development, in the distal AEC (39).

Developmental biology: order in the lung. Nature 2008; 453:733–735. 8. Unbekandt M, del Moral PM, Sala FG, et al. Tracheal occlusion increases the rate of epithelial branching of embryonic mouse lung via the FGF10-FGFR2b-Sprouty2 pathway. Mech Dev 2008; 125:314–324. 9. Hashimoto S, Nakano H, Singh G, et al. Expression of Spred and Sprouty in developing rat lung. Mech Dev 2002; 119(suppl 1):S303–S309. 10. Lu MM, Li S, Yang H, et al. Foxp4: a novel member of the Foxp subfamily of winged-helix genes co-expressed with Foxp1 and Foxp2 in pulmonary and gut tissues.

Newer studies in mice show that oxygen-induced BPD models can be ameliorated in terms of alveolarization both by VEGF therapy and by various kinds of exogenous stem cells. Thus, hope springs eternal for improved postnatal lung recovery from the ravages of prematurity, oxygen plus pressure plus time. Translation of this hope into practices that can prevent or improve BPD outcomes may depend on an increasingly thorough integrative understanding of the genetics of lung development, injury, repair, and regeneration in the human premature lung.

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